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First, regulation of the partitioning of arginine between iNOS and Arg1 has been shown in the arthritis setting to alter the inflammatory output of tissue macrophages and affect disease phenotypes (122), suggesting that the “arginine pathway node-synovial macrophage function-arthritis phenotype” axis is amenable to intervention

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HyperChIP: identification of hypervariable signals across ChIP-seq or ATAC-seq samples